The quantity and quality of one's social relationships have been reliably related to morbidity and mortality. A significant issue concerns the potential mechanisms linking social relationships to such long-term health consequence. Our program of research has been aimed at examining the autonomic, endocrine, and immune systems as potential physiological pathways by which social relationships influence adaptation to stress and long-term physical health.
We have proposed a more comprehensive framework that specifies the natural heterogeneity that exists in social networks. This research suggest the utility of separating network ties who are purely positive (i.e., supportive) and network members that are a source of both positive and negative interactions (i.e., ambivalence - competitive friend, volatile romance). In fact, our research suggest that such ambivalent network ties are associated with detrimental influences on cardiovascular function (see refs). Our current work is specifying the pathways responsible for these associations at different levels of analysis (e.g., social-cognitive).
A cross-cutting interest in my program of research has been on modeling integrative stress-induced biological mechanisms. This work has informed my research highlighted above on relationships and health, as well as related work on age-related differences in physiological reactivity to stress.
The research described here was generously supported by the National Institute of Aging, the National Institute of Mental Health, and the National Heart, Lung, and Blood Institute.